1996 112:1150–60.īrown JW, Ruzmetov M, Okada Y, Vijay P, Rodefeld MD, Turrentine MW. Repair of interrupted aortic arch: a ten-year experience. Serraf A, Lacour-Gayet F, Robotin M, Bruniaux J, Sousa-Uva M, Roussin R, Planche C. Necrotizing enterocolitis in neonates with congenital heart disease: risk factors and outcomes. McElhinney DB, Hedrick HL, Bush DM, Pereira GR, Stafford PW, Gaynor JW, Spray TL, Wernovsky G. Interruption of aortic arch and hypoplastic left heart syndrome. 2005 8:92–102.ĭevloo-Blancquaert A, Titus JL, Edwards JE, Vallaeys JH, De Gezelle HR, Coppens M. Semin Thorac Cardiovasc Surg Pediatr Card Surg Annu. Interrupted aortic arch: surgical decision making. Tchervenkov CI, Jacobs JP, Sharma K, Ungerleider RM. The management of severe subaortic stenosis, ventricular septal defect, and aortic arch obstruction in the neonate. 1993 22:1953–60.īove EL, Minich LL, Pridjian AK, Lupinetti FM, Snider AR, Dick M, Beekman III RH. Echocardiographic predictors of left ventricular outflow tract obstruction after repair of interrupted aortic arch. Geva T, Hornberger LK, Sanders SP, Jonas RA, Ott DA, Colan SD. Does left ventricular outflow tract obstruction influence outcome of interrupted aortic arch repair? Annals of Thoracic Surgery. 1976 37:78–81.įulton JO, Mas C, Brizard CP, Cochrane AD, Karl TR. Anterolateral muscle bundle of the left ventricle, bulboventricular flange and subaortic stenosis. Predictors of left ventricular outflow obstruction following single-stage repair of interrupted aortic arch and ventricular septal defect. Salem MM, Starnes VA, Wells WJ, Acherman RJ, Chang RK, Luciani GB, Wong PC. Deletion 22q11 in patients with interrupted aortic arch. Marino B, Digilio MC, Persiani M, Di Donato R, Toscano A, Giannotti A, Dallapiccola B. Frequency of 22q11 deletions in patients with conotruncal defects. Goldmuntz E, Clark BJ, Mitchell LE, Jawad AF, Cuneo BF, Reed L, McDonald-McGinn D, Chien P, Feuer J, Zackai EH, Emanuel BS, Driscoll DA. Incidence and significance of 22q11.2 hemizygosity in patients with interrupted aortic arch. Rauch A, Hofbeck M, Leipold G, Klinge J, Trautmann U, Kirsch M, Singer H, Pfeiffer RA. A genetic etiology for interruption of the aortic arch type B. Lewin MB, Lindsay EA, Jurecic V, Goytia V, Towbin JA, Baldini A. Cardiovascular anomalies in DiGeorge syndrome and importance of neural crest as a possible pathogenetic factor. Association of interrupted aortic arch with malformations producing reduced blood flow to the fourth aortic arches. Hemodynamic considerations in the development of narrowing of the aorta. Interruption of the aortic arch and coarctation of the aorta: pathogenetic relations. The ductus arterious and associated cardiac anomalies in interruption of the aortic arch. Oppenheimer-Dekker A, Gittenberger-de Groot AC, Roozendaal H. Solitary interruption of the arch of the aorta. Risk factors associated with mortality and interventions in 472 neonates with interrupted aortic arch: a Congenital Heart Surgeons Society study. McCrindle BW, Tchervenkov CI, Konstantinov IE, Williams WG, Neirotti RA, Jacobs ML, Blackstone EH. Congenital Heart Surgery Nomenclature and Database Project: patent ductus arteriosus, coarctation of the aorta, interrupted aortic arch. Anatomic features affect mortality and initial LVOT procedures, whereas characteristics of the arch repair affect arch reintervention. Recurrence of arch obstruction has been reported from 10 to 30 %, while recurrence of LVOTO has averaged 20 %. Current early mortality for repair of IAA with VSD ranges from 4 to 10 %. A severe LVOTO may require more aggressive procedures, e.g. Aortic arch reconstruction is accomplished by end-to-side anastomosis with the ascending aorta, more or less associated with patch augmentation of the anastomosis. Staged repair was popular in the past, but primary repair through a median sternotomy (using either deep hypothermic circulatory arrest or moderately hypothermic antegrade selective cerebral perfusion) is currently the preferred approach. DiGeorge syndrome is another frequently associated feature with important clinical implications. Careful preoperative assessment, and particularly the identification of a severe left ventricular outflow tract obstruction (LVOTO, <4 mm), is crucial for a successful outcome. The critical condition of these neonates dictates early referral and surgical treatment. Only type B is, but not exclusively, a true cono-truncal anomaly. Three types of IAA – A, B and C – are classicaly recognized. Interrupted aortic arch (IAA), usually associated with a ventricular septal defect (VSD), is a rapidly fatal congenital heart anomaly with ductus-dependent systemic circulation.
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